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max in WT synaptoneurosomes, suggesting that Src signaling may be downregulated in KI synapses. However, our power to rescue SERT functionality in KI midbrain synaptoneurosomes because of the inhibition of FAK implies elevated FAK signaling downstream of your Pro32Pro33 mutant, as verified by enhanced pFAK localization in 5-HT synapses. Our details